Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA demethylation in G1 phase-arrested and proliferative HeLa cells

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Last updated 12 maio 2024
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
DNA demethylation by 5-aza-2′-deoxycytidine is imprinted, targeted to euchromatin, and has limited transcriptional consequences, Epigenetics & Chromatin
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
The 10 most upregulated development-related genes after 5-Aza-dC
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
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Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
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Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
Gene expression of DNMT1, TET1 and TET2 in VPA- and 5-aza-CdR- treated
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
Sodium Valproate Modulates the Methylation Status of Lysine Residues 4, 9 and 27 in Histone H3 of HeLa Cells
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
Transcriptome and methylome changes in U937 cells upon treatment with
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
Frontiers Resistance to Hypomethylating Agents in Myelodysplastic Syndrome and Acute Myeloid Leukemia From Clinical Data and Molecular Mechanism
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
Valproic acid influences the expression of genes implicated with hyperglycaemia-induced complement and coagulation pathways. - Abstract - Europe PMC
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
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Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
Nutrients, Free Full-Text
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
DNA demethylation by 5-aza-2′-deoxycytidine is imprinted, targeted to euchromatin, and has limited transcriptional consequences, Epigenetics & Chromatin
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
DNA demethylation by 5-aza-2′-deoxycytidine is imprinted, targeted to euchromatin, and has limited transcriptional consequences, Epigenetics & Chromatin
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
The hypomethylating agent Decitabine causes a paradoxical increase in 5-hydroxymethylcytosine in human leukemia cells
Sodium valproate and 5-aza-2′-deoxycytidine differentially modulate DNA  demethylation in G1 phase-arrested and proliferative HeLa cells
Expression of Dppa2 and Dppa4 coincides with promoter DNA

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